It does not have to be Lyme!! Treat these infections similarly, although we may not know the name of all of them yet. Lowering the total body burden of pathogens is vital to success in most of these chronic, often auto-immune related conditions.
I like Oxidative Medicine and UVB with Ozone but since everyone is not into that please learn to aggressively use ACS 200, 1 ounce to load and often ½ ounce twice a day for a week or a month depending on response. Of course, IV Ascorbic Acid helped by BIOE’NR-G’Y C that really MAINTAINS higher intracellular activity than any other Vitamin C available anywhere is a great way to offer help. I strongly recommend learning what is in Immuni-T-2, three caps bid to aid the body in keeping the pathogen load down.
Then learn about the exciting NEW Zeolite based detox formulas now available through Longevity Plus, ZeoGold and BioE’nR-G’y C Zeolite, as research is showing that the better Zeolite products clearly improve immune function. This should be obvious since GALT, the gut associated lymphoid tissue with Peyer’s patches etc, is relieved of toxins by keeping Zeolite in the intestine. Since this works for air filters and water filters, there is no question that the superior Zeolite products now available through Longevity Plus will offer substantial health improvements in patients since we all have toxins in our Intestine at all times, and binding those toxins is being shown to be very useful. Furthermore, Zeolite handles AFLOTOXINS and AMMONIA and many other toxins that standard chelators are not affecting. Preliminary data also suggests that the improved nutrient utilization widely documented in Zeolite research may relate to BIOFILM effects.
Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
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From: INTEGRATIVE MEDICAL-CONSULTING
Chlamydia May Play Role In A Type Of Arthritis
ScienceDaily (May 6, 2009) — Spondylarthritis (SpA) represents a group of arthritidies that share clinical features such as inflammatory back pain and inflammation at sites where tendons attach to bone. It includes ankylosing spondylitis (AS), psoriatic arthritis, inflammatory bowel-disease-related arthritis, reactive arthritis (ReA) and undifferentiated spondylarthritides (uSpA).
Since Chlamydia trachomatis or Chlamydia pneumoniae (which are often asymptomatic) frequently cause ReA, a new study examined whether there was a connection between these two infections and uSpA.
Led by John D. Carter of theUniversity of South Florida, the study involved blood and synovial tissue analysis from 26 patients who had chronic uSpA or Chlamydia-induced ReA. Synovial tissue samples from 167 osteoarthritis patients were used as controls. Samples were analyzed to assess chlamydial DNA and the 26 subjects were asked if they had any known exposure to Chlamydia trachomatis or Chlamydia pneumoniae and if so, the infection was documented in relation to the onset of their uSpA. They also underwent a physical exam that included evaluation of swollen and tender joints and other symptoms of SpA. The results showed that the rate of Chlamydia infection was 62 percent in uSpA patients, significantly higher than the 12 percent seen in control subjects.
It is believed that as many as 150,000 cases of Chlamydia trachomatis-induced ReA may appear in the U.S. each year compared to about 125,000 new cases of rheumatoid arthritis. This is a low estimate since it does not include cases resulting from Chlamydia pneumoniae. “Thus, Chlamydia-induced ReA represents a considerable burden on the health care systems of the U.S. and other nations, and its impact on those systems may well be significantly underrecognized,” the authors state.
Most women with genital Chlamydia trachomatis infection have no symptoms at the time of the initial infection; this was also true of the patients in the study who had DNA evidence of Chlamydia. For Chlamydia pneumoniae, as many as 70 percent of acute infections are asymptomatic and, even when there are symptoms, definitive identification of the organism is rare. The authors point out that relying on identification of a symptomatic infection may therefore result in routine underdiagnosis or misdiagnosis of Chlamydia-induced ReA.
They add that because ReA is a type of SpA and patients with ReA do not present with the classic combination of symptoms of arthritis, conjunctivitis/iritis and urethritis, it is reasonable to believe that Chlamydia trachomatis plays a role in causing uSpA, which may in fact be ReA. They conclude that although there is no diagnostic test for Chlamydia-induced ReA, testing for chlamydial DNA in the synovial tissue of patients thought to have ReA may be the most accurate way of diagnosing the condition.
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Journal reference:
1. Carter et al. Chlamydiae as etiologic agents in chronic undifferentiated spondylarthritis. Arthritis & Rheumatism, 2009; 60 (5): 1311 DOI: 10.1002/art.24431
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